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Environmental Factors in the Causation of Autism
Wednesday, November 12, 2003 (19:06:43)
Posted by Sylvia
Paper by Paul Shattock
Introduction
It is universally accepted that there is a considerable genetic
element in the incidence of autism. The occurrence of so many
families where multi-incidences of autism and related spectral
disorders exist is compelling evidence.
However, this is probably
not the whole story. Even the strongest proponents of genetics
research are now beginning to talk in terms of "genetic fragility or
predisposition". If, as seems highly possible, the incidence of
autism is increasing, then factors other than the purely genetic
must be implicated.
Increasing reported levels of autism have
been reported from many parts of the world but, as has been
pointed out on a number of occasions, this may reflect no more
that increasing awareness of the disorder and changing diagnostic
criteria (Gillberg 1998a; Wing 1996).
However, two recent reports (California (1999); Taylor (1999))
have highlighted what would appear to be very significant
increases in the incidence. It must be remarked that the latter
was intended to demonstrate that the introduction of the
combined Measles, Mumps and Rubella (MMR) vaccine was, in no
way, a factor in this increase.
Many of those who actually read this
paper were unable to reach the same conclusions as the authors
but, even so, there was a dramatic (1,700%?) increase of
reported incidence between 1979 and 1992 (birth dates). The
California (1999) report showed a 273% increase, in the State of
California over a similar (but not identical) period of time.
Informal and unpublished data from many parts of the UK and
other parts of the world are showing similar increases. It is
alleged that parallel, staggering increases in incidence are
occurring in similar disorders such as dyslexia; Attention Deficit
Disorder (ADD) which may be combined with hyperactivity (ADHD).
Indeed, reports from the US and Australia suggest that 10% of
children of school age are currently taking Ritalin to ameliorate
the symptoms of ADHD. In other circumstances, these increases
would be regarded as an epidemic and worthy of considerable
research but, so far, environmental factors have been completely
and utterly ignored by governmentally funded agencies within the
UK.
If these increases are, indeed, genuine then there must be
factors, additional to the purely genetic, which are involved and
which may trigger the problems.
It may be of interest and relevance to examine other diseases or
disorders which have, in comparatively recent history, increased or
decreased markedly in incidence. There could be lessons which
can be learned and which could be used to explain the apparent
upsurge in incidence of the disorders described above.
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